microarray-based comparative genomic hybridization (acgh) 180k Search Results


kit nucleospin blood l  (MACHEREY NAGEL)
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MACHEREY NAGEL kit nucleospin blood l
Kit Nucleospin Blood L, supplied by MACHEREY NAGEL, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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sureprint g3 cgh 4 × 180k microarrays  (Agilent technologies)
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Agilent technologies sureprint g3 cgh 4 × 180k microarrays
Sureprint G3 Cgh 4 × 180k Microarrays, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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4×180k sureprint g3 human cgh microarray  (Agilent technologies)
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Agilent technologies 4×180k sureprint g3 human cgh microarray
( a ) Overview and design of the experimental procedures. ( b ) Illustration of targeted 16p11.2 rMDS segment and/or SDs. For simplicity, only protein coding genes are shown from the Ensembl GRCh37 V.71 annotation . The targeted unique genomic segment for the dual-guide 575 kb deletion is indicated in yellow, and the single-guide RNA targeting the SDs to promote a model of NAHR-mediated CNV is indicated in red (Target sequence: 5′-ACATGCCTATATCGCATAG -3′, chromosome 16: 29,487,572–29,487,590 and 30,226,917–30,226,935). ( c ) Efficiency of CRISPR/Cas9 generation of the 740 kb rMDS using the single-guide SCORE method that targets the SDs is shown, as determined by copy number screening assay for six genes. Further characterization by <t>microarray</t> and RNAseq was performed for a subset of microdeletion clones (6), and all microduplication clones (5). ( d ) Microarray analyses showing deletion (CRISPR Del) and duplication (CRISPR Dup) of the 16p11.2 region in CRISPR-treated lines is shown. Gains or losses of 16p11.2 region were determined by normalized log2 ratios. No off-target CNVs were observed in CRISPR-generated clones.
4×180k Sureprint G3 Human Cgh Microarray, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/4×180k sureprint g3 human cgh microarray/product/Agilent technologies
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4×180k sureprint g3 human cgh microarray - by Bioz Stars, 2026-04
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180k sureprint g3 cgh microarray  (Agilent technologies)
90
Agilent technologies 180k sureprint g3 cgh microarray
( a ) Overview and design of the experimental procedures. ( b ) Illustration of targeted 16p11.2 rMDS segment and/or SDs. For simplicity, only protein coding genes are shown from the Ensembl GRCh37 V.71 annotation . The targeted unique genomic segment for the dual-guide 575 kb deletion is indicated in yellow, and the single-guide RNA targeting the SDs to promote a model of NAHR-mediated CNV is indicated in red (Target sequence: 5′-ACATGCCTATATCGCATAG -3′, chromosome 16: 29,487,572–29,487,590 and 30,226,917–30,226,935). ( c ) Efficiency of CRISPR/Cas9 generation of the 740 kb rMDS using the single-guide SCORE method that targets the SDs is shown, as determined by copy number screening assay for six genes. Further characterization by <t>microarray</t> and RNAseq was performed for a subset of microdeletion clones (6), and all microduplication clones (5). ( d ) Microarray analyses showing deletion (CRISPR Del) and duplication (CRISPR Dup) of the 16p11.2 region in CRISPR-treated lines is shown. Gains or losses of 16p11.2 region were determined by normalized log2 ratios. No off-target CNVs were observed in CRISPR-generated clones.
180k Sureprint G3 Cgh Microarray, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/180k sureprint g3 cgh microarray/product/Agilent technologies
Average 90 stars, based on 1 article reviews
180k sureprint g3 cgh microarray - by Bioz Stars, 2026-04
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uls labeling  (Agilent technologies)
90
Agilent technologies uls labeling
( a ) Overview and design of the experimental procedures. ( b ) Illustration of targeted 16p11.2 rMDS segment and/or SDs. For simplicity, only protein coding genes are shown from the Ensembl GRCh37 V.71 annotation . The targeted unique genomic segment for the dual-guide 575 kb deletion is indicated in yellow, and the single-guide RNA targeting the SDs to promote a model of NAHR-mediated CNV is indicated in red (Target sequence: 5′-ACATGCCTATATCGCATAG -3′, chromosome 16: 29,487,572–29,487,590 and 30,226,917–30,226,935). ( c ) Efficiency of CRISPR/Cas9 generation of the 740 kb rMDS using the single-guide SCORE method that targets the SDs is shown, as determined by copy number screening assay for six genes. Further characterization by <t>microarray</t> and RNAseq was performed for a subset of microdeletion clones (6), and all microduplication clones (5). ( d ) Microarray analyses showing deletion (CRISPR Del) and duplication (CRISPR Dup) of the 16p11.2 region in CRISPR-treated lines is shown. Gains or losses of 16p11.2 region were determined by normalized log2 ratios. No off-target CNVs were observed in CRISPR-generated clones.
Uls Labeling, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/uls labeling/product/Agilent technologies
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uls labeling - by Bioz Stars, 2026-04
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sureprint g3 human cgh+snp 4 × 180k microarray kit  (Agilent technologies)
90
Agilent technologies sureprint g3 human cgh+snp 4 × 180k microarray kit
( a ) Overview and design of the experimental procedures. ( b ) Illustration of targeted 16p11.2 rMDS segment and/or SDs. For simplicity, only protein coding genes are shown from the Ensembl GRCh37 V.71 annotation . The targeted unique genomic segment for the dual-guide 575 kb deletion is indicated in yellow, and the single-guide RNA targeting the SDs to promote a model of NAHR-mediated CNV is indicated in red (Target sequence: 5′-ACATGCCTATATCGCATAG -3′, chromosome 16: 29,487,572–29,487,590 and 30,226,917–30,226,935). ( c ) Efficiency of CRISPR/Cas9 generation of the 740 kb rMDS using the single-guide SCORE method that targets the SDs is shown, as determined by copy number screening assay for six genes. Further characterization by <t>microarray</t> and RNAseq was performed for a subset of microdeletion clones (6), and all microduplication clones (5). ( d ) Microarray analyses showing deletion (CRISPR Del) and duplication (CRISPR Dup) of the 16p11.2 region in CRISPR-treated lines is shown. Gains or losses of 16p11.2 region were determined by normalized log2 ratios. No off-target CNVs were observed in CRISPR-generated clones.
Sureprint G3 Human Cgh+Snp 4 × 180k Microarray Kit, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/sureprint g3 human cgh+snp 4 × 180k microarray kit/product/Agilent technologies
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400k sureprint g3 cgh microarray  (Agilent technologies)
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Agilent technologies 400k sureprint g3 cgh microarray
( a ) Overview and design of the experimental procedures. ( b ) Illustration of targeted 16p11.2 rMDS segment and/or SDs. For simplicity, only protein coding genes are shown from the Ensembl GRCh37 V.71 annotation . The targeted unique genomic segment for the dual-guide 575 kb deletion is indicated in yellow, and the single-guide RNA targeting the SDs to promote a model of NAHR-mediated CNV is indicated in red (Target sequence: 5′-ACATGCCTATATCGCATAG -3′, chromosome 16: 29,487,572–29,487,590 and 30,226,917–30,226,935). ( c ) Efficiency of CRISPR/Cas9 generation of the 740 kb rMDS using the single-guide SCORE method that targets the SDs is shown, as determined by copy number screening assay for six genes. Further characterization by <t>microarray</t> and RNAseq was performed for a subset of microdeletion clones (6), and all microduplication clones (5). ( d ) Microarray analyses showing deletion (CRISPR Del) and duplication (CRISPR Dup) of the 16p11.2 region in CRISPR-treated lines is shown. Gains or losses of 16p11.2 region were determined by normalized log2 ratios. No off-target CNVs were observed in CRISPR-generated clones.
400k Sureprint G3 Cgh Microarray, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/400k sureprint g3 cgh microarray/product/Agilent technologies
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180k cytosure isca v2 array  (Oxford Gene Technology)
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Oxford Gene Technology 180k cytosure isca v2 array
( a ) Overview and design of the experimental procedures. ( b ) Illustration of targeted 16p11.2 rMDS segment and/or SDs. For simplicity, only protein coding genes are shown from the Ensembl GRCh37 V.71 annotation . The targeted unique genomic segment for the dual-guide 575 kb deletion is indicated in yellow, and the single-guide RNA targeting the SDs to promote a model of NAHR-mediated CNV is indicated in red (Target sequence: 5′-ACATGCCTATATCGCATAG -3′, chromosome 16: 29,487,572–29,487,590 and 30,226,917–30,226,935). ( c ) Efficiency of CRISPR/Cas9 generation of the 740 kb rMDS using the single-guide SCORE method that targets the SDs is shown, as determined by copy number screening assay for six genes. Further characterization by <t>microarray</t> and RNAseq was performed for a subset of microdeletion clones (6), and all microduplication clones (5). ( d ) Microarray analyses showing deletion (CRISPR Del) and duplication (CRISPR Dup) of the 16p11.2 region in CRISPR-treated lines is shown. Gains or losses of 16p11.2 region were determined by normalized log2 ratios. No off-target CNVs were observed in CRISPR-generated clones.
180k Cytosure Isca V2 Array, supplied by Oxford Gene Technology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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sureprint g3 rat cgh 4 × 180k microarray, g4826a#27064  (Agilent technologies)
90
Agilent technologies sureprint g3 rat cgh 4 × 180k microarray, g4826a#27064
Expression <t>microarray</t> analysis at the subacute phase of Fe-NTA-induced renal carcinogenesis sorts out mitochondrial and iron metabolisms as candidate responsible pathways in Brca1 MUT(L63X/+) rat. (A) Results of hierarchical clustering and GO term analysis for the genes differentially expressed between WT and Brca1 MUT at 3 weeks in the Fe-NTA-induced renal carcinogenesis protocol (refer to ). (B) Renal Brca1 expression during the subacute phase of Fe-NTA-induced renal carcinogenesis protocol with qPCR (left panel; means ± SD) and immunoblot analysis (right panels). (C) Examples of altered metabolisms and mitochondrial dysfunction with expression microarray analysis specifically in the kidney of Brca1 MUT rats during the subacute phase of Fe-NTA-induced renal carcinogenesis (n = 3; * P < 0.05, ** P < 0.01, *** P < 0.001 vs wild-type Fe-NTA renal carcinogenesis protocol at 3 weeks). Refer to text for details.
Sureprint G3 Rat Cgh 4 × 180k Microarray, G4826a#27064, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/sureprint g3 rat cgh 4 × 180k microarray, g4826a#27064/product/Agilent technologies
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Image Search Results


( a ) Overview and design of the experimental procedures. ( b ) Illustration of targeted 16p11.2 rMDS segment and/or SDs. For simplicity, only protein coding genes are shown from the Ensembl GRCh37 V.71 annotation . The targeted unique genomic segment for the dual-guide 575 kb deletion is indicated in yellow, and the single-guide RNA targeting the SDs to promote a model of NAHR-mediated CNV is indicated in red (Target sequence: 5′-ACATGCCTATATCGCATAG -3′, chromosome 16: 29,487,572–29,487,590 and 30,226,917–30,226,935). ( c ) Efficiency of CRISPR/Cas9 generation of the 740 kb rMDS using the single-guide SCORE method that targets the SDs is shown, as determined by copy number screening assay for six genes. Further characterization by microarray and RNAseq was performed for a subset of microdeletion clones (6), and all microduplication clones (5). ( d ) Microarray analyses showing deletion (CRISPR Del) and duplication (CRISPR Dup) of the 16p11.2 region in CRISPR-treated lines is shown. Gains or losses of 16p11.2 region were determined by normalized log2 ratios. No off-target CNVs were observed in CRISPR-generated clones.

Journal: Nature neuroscience

Article Title: Engineering microdeletions and microduplications by targeting segmental duplications with CRISPR

doi: 10.1038/nn.4235

Figure Lengend Snippet: ( a ) Overview and design of the experimental procedures. ( b ) Illustration of targeted 16p11.2 rMDS segment and/or SDs. For simplicity, only protein coding genes are shown from the Ensembl GRCh37 V.71 annotation . The targeted unique genomic segment for the dual-guide 575 kb deletion is indicated in yellow, and the single-guide RNA targeting the SDs to promote a model of NAHR-mediated CNV is indicated in red (Target sequence: 5′-ACATGCCTATATCGCATAG -3′, chromosome 16: 29,487,572–29,487,590 and 30,226,917–30,226,935). ( c ) Efficiency of CRISPR/Cas9 generation of the 740 kb rMDS using the single-guide SCORE method that targets the SDs is shown, as determined by copy number screening assay for six genes. Further characterization by microarray and RNAseq was performed for a subset of microdeletion clones (6), and all microduplication clones (5). ( d ) Microarray analyses showing deletion (CRISPR Del) and duplication (CRISPR Dup) of the 16p11.2 region in CRISPR-treated lines is shown. Gains or losses of 16p11.2 region were determined by normalized log2 ratios. No off-target CNVs were observed in CRISPR-generated clones.

Article Snippet: Array-based comparative genomic hybridization (aCGH) was performed on the Agilent 4×180K SurePrint G3 Human CGH Microarray (Design #022060) according to the protocol provided by the manufacturer.

Techniques: Sequencing, CRISPR, Screening Assay, Microarray, Clone Assay, Generated

Expression microarray analysis at the subacute phase of Fe-NTA-induced renal carcinogenesis sorts out mitochondrial and iron metabolisms as candidate responsible pathways in Brca1 MUT(L63X/+) rat. (A) Results of hierarchical clustering and GO term analysis for the genes differentially expressed between WT and Brca1 MUT at 3 weeks in the Fe-NTA-induced renal carcinogenesis protocol (refer to ). (B) Renal Brca1 expression during the subacute phase of Fe-NTA-induced renal carcinogenesis protocol with qPCR (left panel; means ± SD) and immunoblot analysis (right panels). (C) Examples of altered metabolisms and mitochondrial dysfunction with expression microarray analysis specifically in the kidney of Brca1 MUT rats during the subacute phase of Fe-NTA-induced renal carcinogenesis (n = 3; * P < 0.05, ** P < 0.01, *** P < 0.001 vs wild-type Fe-NTA renal carcinogenesis protocol at 3 weeks). Refer to text for details.

Journal: Redox Biology

Article Title: BRCA1 haploinsufficiency promotes chromosomal amplification under Fenton reaction-based carcinogenesis through ferroptosis-resistance

doi: 10.1016/j.redox.2022.102356

Figure Lengend Snippet: Expression microarray analysis at the subacute phase of Fe-NTA-induced renal carcinogenesis sorts out mitochondrial and iron metabolisms as candidate responsible pathways in Brca1 MUT(L63X/+) rat. (A) Results of hierarchical clustering and GO term analysis for the genes differentially expressed between WT and Brca1 MUT at 3 weeks in the Fe-NTA-induced renal carcinogenesis protocol (refer to ). (B) Renal Brca1 expression during the subacute phase of Fe-NTA-induced renal carcinogenesis protocol with qPCR (left panel; means ± SD) and immunoblot analysis (right panels). (C) Examples of altered metabolisms and mitochondrial dysfunction with expression microarray analysis specifically in the kidney of Brca1 MUT rats during the subacute phase of Fe-NTA-induced renal carcinogenesis (n = 3; * P < 0.05, ** P < 0.01, *** P < 0.001 vs wild-type Fe-NTA renal carcinogenesis protocol at 3 weeks). Refer to text for details.

Article Snippet: We labeled DNA from 16 rat primary RCCs (4 in wild-type without metastasis, 4 in wild-type with pulmonary metastasis, 4 in Brca1-MUT rat without metastasis, and 4 in Brca1-MUT rat with pulmonary metastasis; randomly selected from RCC samples of appropriate size [diameter >15 mm] without massive necrosis and obtained by scheduled autopsy; ) with Cy-5 and the corresponding control DNA with Cy-3, which were applied to the aCGH microarray slides (SurePrint G3 Rat CGH 4 × 180k Microarray, G4826A#27064, Agilent Technologies, Santa Clara, CA) according to the Agilent Oligonucleotide Array-Based CGH for Genomic DNA Analysis protocol Ver.7.3.

Techniques: Expressing, Microarray, Western Blot